Suppression of Cpn10 Increases Mitochondrial Fission and Dysfunction in Neuroblastoma Cells-Reference-Cited by-同舟云学术

Suppression of Cpn10 Increases Mitochondrial Fission and Dysfunction in Neuroblastoma Cells

Published:2014-11-12 Issue:11 Volume:9 Page:e112130
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Park So Jung,Jo Doo Sin,Shin Ji Hyun,Kim Eun Sung,Jo Yoon Kyung,Choi Eun Sun,Seo Hae Mi,Kim Sung Hyun,Hwang Jung Jin,Jo Dong-Gyu,Koh Jae-Young,Cho Dong-Hyung

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference49 articles.

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1. Inhibition of mitochondrial translocase SLC25A5 and histone deacetylation is an effective combination therapy in neuroblastoma;International Journal of Cancer;2022-11-17

2. Neuroblastoma—A Neural Crest Derived Embryonal Malignancy;Frontiers in Molecular Neuroscience;2019-01-29

3. Molecular mechanisms and therapeutic targets in neuroblastoma;Pharmacological Research;2018-05

4. Heterogeneous nuclear ribonucleoprotein A1 post-transcriptionally regulates Drp1 expression in neuroblastoma cells;Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms;2015-12

5. Interaction of Heat Shock Protein Cpn10 with the Cyclin E/Cdk2 Substrate Nuclear Protein Ataxia-Telangiectasia (NPAT) Is Involved in Regulating Histone Transcription;Journal of Biological Chemistry;2015-12