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Lack of Renoprotective Effect of Chronic Intravenous Angiotensin-(1-7) or Angiotensin-(2-10) in a Rat Model of Focal Segmental Glomerulosclerosis

  • Published:2014-10-22 Issue:10 Volume:9 Page:e110083
  • ISSN:1932-6203
  • Container-title:PLoS ONE
  • language:en
  • Short-container-title:PLoS ONE

Author:

Velez Juan Carlos Q.,Janech Michael G.,Hicks Megan P.,Morinelli Thomas A.,Rodgers Jessalyn,Self Sally E.,Arthur John M.,Fitzgibbon Wayne R.

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference46 articles.

1. Role of the renin-angiotensin-aldosterone system in the progression of renal disease: a critical review;HN Ibrahim;Seminars in nephrology,1997

2. Renin-angiotensin system may trigger kidney damage in NOD mice;JA Colucci;Journal of the renin-angiotensin-aldosterone system: JRAAS,2011

3. Angiotensin II-dependent persistent podocyte loss from destabilized glomeruli causes progression of end stage kidney disease;A Fukuda;Kidney international,2012

4. Angiotensin I is largely converted to angiotensin (1-7) and angiotensin (2-10) by isolated rat glomeruli;JC Velez;Hypertension,2009

5. Characterization of renin-angiotensin system enzyme activities in cultured mouse podocytes;JC Velez;American journal of physiology Renal physiology,2007

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