Loss of Pol32 in Drosophila melanogaster Causes Chromosome Instability and Suppresses Variegation-Reference-Cited by-同舟云学术

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Loss of Pol32 in Drosophila melanogaster Causes Chromosome Instability and Suppresses Variegation

Published:2015-03-31 Issue:3 Volume:10 Page:e0120859
ISSN:1932-6203
Container-title:PLOS ONE
language:en
Short-container-title:PLoS ONE

Author:

Tritto Patrizia,Palumbo Valeria,Micale Lucia,Marzulli Marco,Bozzetti Maria Pia,Specchia Valeria,Palumbo Gioacchino,Pimpinelli Sergio,Berloco Maria

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference66 articles.

1. Structure of DNA polymerase delta from Saccharomyces cerevisiae;E Johansson;J Biol Chem,2001

2. DNA polymerase and switch by sharing accessory subunits of DNA polymerase;AG Baranovskiy;J. Biol. Chem.,2012

3. Pol31 and Pol32 subunits of yeast DNA polymerase delta are also essential subunits of DNA polymerase zeta;RE Johnson;Proc Natl Acad Sci U S A,2012

4. A four-subunit DNA polymerase zeta complex containing Pol delta accessory subunits is essential for PCNA-mediated mutagenesis;AV Makarova;Nucleic Acids Res,2012

5. Subunit sharing among high- and low-fidelity DNA polymerases;LD Langston;Proc Natl Acad Sci U S A,2012

Cited by 8 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Association of Mutations in Replicative DNA Polymerase Genes with Human Disease: Possible Application of Drosophila Models for Studies;International Journal of Molecular Sciences;2023-04-29

2. The DNA polymerases of Drosophila melanogaster;Fly;2020-01-14

3. Drosophila Morgana is an Hsp90-interacting protein with a direct role in microtubule polymerization;Journal of Cell Science;2020-01-01

4. The Drosophila melanogaster PIF1 Helicase Promotes Survival During Replication Stress and Processive DNA Synthesis During Double-Strand Gap Repair;Genetics;2019-11-01

5. The processivity factor Pol32 mediates nuclear localization of DNA polymerase delta and prevents chromosomal fragile site formation in Drosophila development;PLOS Genetics;2019-05-17

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