Gene Expression Analyses during Spontaneous Reversal of Cardiomyopathy in Mice with Repressed Nuclear CUG-BP, Elav-Like Family (CELF) Activity in Heart Muscle
Author:
Publisher
Public Library of Science (PLoS)
Subject
Multidisciplinary
Reference47 articles.
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2. Transgenic mice expressing CUG-BP1 reproduce splicing mis-regulation observed in myotonic dystrophy;TH Ho;Hum Mol Genet,2005
3. Heart-specific overexpression of CUGBP1 reproduces functional and molecular abnormalities of myotonic dystrophy type 1;M Koshelev;Hum Mol Genet,2010
4. Cardiac-specific repression of CELF activity disrupts alternative splicing and causes cardiomyopathy;A Ladd;Mol Cell Biol,2005
5. Disruption of splicing regulated by a CUG-binding protein in myotonic dystrophy;A Philips;Science,1998
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3. Neonatal cardiac dysfunction and transcriptome changes caused by the absence of Celf1;Scientific Reports;2016-10-19
4. Identification of Targets of CUG-BP, Elav-Like Family Member 1 (CELF1) Regulation in Embryonic Heart Muscle;PLOS ONE;2016-02-11
5. New Insights Into the Role of RNA-Binding Proteins in the Regulation of Heart Development;International Review of Cell and Molecular Biology;2016
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