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Neurodegeneration and Epilepsy in a Zebrafish Model of CLN3 Disease (Batten Disease)

  • Published:2016-06-21 Issue:6 Volume:11 Page:e0157365
  • ISSN:1932-6203
  • Container-title:PLOS ONE
  • language:en
  • Short-container-title:PLoS ONE

Author:

Wager Kim,Zdebik Anselm A.,Fu Sonia,Cooper Jonathan D.,Harvey Robert J.,Russell ClaireORCID

Funder

Batten Disease Support and Research Association

Batten Disease Family Association

University of London

Newlife Foundation for Disabled Children

Royal Society

Bloomsbury Consortium

Peter Samuel Trust Fund

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference54 articles.

1. The epidemiology of progressive intellectual and neurological deterioration in childhood;C Verity;Arch Dis Child,2010

2. The neuronal ceroid lipofuscinoses (Batten disease);SE Mole,2011

3. Genetic basis and phenotypic correlations of the neuronal ceroid lipofusinoses;V Warrier;Biochim Biophys Acta,2013

4. Batten disease and the ATP synthase subunit c turnover pathway: raising antibodies to subunit c;DN Palmer;Am J Med Genet,1995

5. Human pathology in NCL;GW Anderson;Biochim Biophys Acta,2013

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1. CLN3 deficiency leads to neurological and metabolic perturbations during early development;Life Science Alliance;2024-01-09

2. Experimental Models of CNS Disorders Related to Lysosomal Storage Diseases;Journal of Evolutionary Biochemistry and Physiology;2023-11

3. Experimental Models of CNS Deficits in Lysosomal Storage Diseases;Российский физиологический журнал им  И  М  Сеченова;2023-11-01

4. Assessing Drug Administration Techniques in Zebrafish Models of Neurological Disease;International Journal of Molecular Sciences;2023-10-04

5. The involvement of Purkinje cells in progressive myoclonic epilepsy: Focus on neuronal ceroid lipofuscinosis;Neurobiology of Disease;2023-09
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