Sar1, a Novel Regulator of ER-Mitochondrial Contact Sites
Author:
Funder
Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung
Universität Basel
Publisher
Public Library of Science (PLoS)
Subject
Multidisciplinary
Reference47 articles.
1. Sar1p N-terminal helix initiates membrane curvature and completes the fission of a COPII vesicle;MC Lee;Cell,2005
2. Regulation of Sar1 NH2 terminus by GTP binding and hydrolysis promotes membrane deformation to control COPII vesicle fission;A Bielli;The Journal of cell biology,2005
3. Sar1 assembly regulates membrane constriction and ER export;KR Long;The Journal of cell biology,2010
4. Insights into the mechanisms of membrane curvature and vesicle scission by the small GTPase Sar1 in the early secretory pathway;H Hariri;Journal of molecular biology,2014
5. An ER-mitochondria tethering complex revealed by a synthetic biology screen;B Kornmann;Science,2009
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1. Activated Drp1 Initiates the Formation of Endoplasmic Reticulum‐Mitochondrial Contacts via Shrm4‐Mediated Actin Bundling;Advanced Science;2023-11
2. COPI-regulated mitochondria-ER contact site formation maintains axonal integrity;Cell Reports;2023-08
3. The alarmone ppGpp selectively inhibits the isoform A of the human small GTPase Sar1;Proteins: Structure, Function, and Bioinformatics;2022-11-21
4. ER exit sites ( ERES ) and ER –mitochondria encounter structures ( ERMES ) often localize proximally;FEBS Letters;2022-10-21
5. Inter-organellar Communication in Parkinson's and Alzheimer's Disease: Looking Beyond Endoplasmic Reticulum-Mitochondria Contact Sites;Frontiers in Neuroscience;2022-06-21
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