Extended Synaptotagmin (ESyt) Triple Knock-Out Mice Are Viable and Fertile without Obvious Endoplasmic Reticulum Dysfunction
Author:
Funder
National Institute of Mental Health
Publisher
Public Library of Science (PLoS)
Subject
Multidisciplinary
Reference48 articles.
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2. Structure and Ca(2)(+)-binding properties of the tandem C(2) domains of E-Syt2;J Xu;Structure,2014
3. PI(4,5)P(2)-dependent and Ca(2+)-regulated ER-PM interactions mediated by the extended synaptotagmins;F Giordano;Cell,2013
4. Three-dimensional architecture of extended synaptotagmin-mediated endoplasmic reticulum-plasma membrane contact sites;R Fernandez-Busnadiego;Proceedings of the National Academy of Sciences of the United States of America,2015
5. Homology of SMP domains to the TULIP superfamily of lipid-binding proteins provides a structural basis for lipid exchange between ER and mitochondria;KO Kopec;Bioinformatics,2010
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1. Correction: Extended Synaptotagmin (ESyt) Triple Knock-Out Mice Are Viable and Fertile without Obvious Endoplasmic Reticulum Dysfunction;PLOS ONE;2024-02-06
2. Stay in touch with the endoplasmic reticulum;Science China Life Sciences;2024-01-04
3. Extended-Synaptotagmin-1 and -2 control T cell signaling and function;EMBO Reports;2023-12-19
4. Enriched Environment Enhances Sociability Through the Promotion of ESyt1-Related Synaptic Formation in the Medial Prefrontal Cortex;Molecular Neurobiology;2023-11-14
5. ESYT1 tethers the ER to mitochondria and is required for mitochondrial lipid and calcium homeostasis;Life Science Alliance;2023-11-06
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