Notch1 Is Not Required for Acinar-to-Ductal Metaplasia in a Model of Kras-Induced Pancreatic Ductal Adenocarcinoma
Author:
Publisher
Public Library of Science (PLoS)
Subject
Multidisciplinary
Reference39 articles.
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3. Preinvasive and invasive ductal pancreatic cancer and its early detection in the mouse;SR Hingorani;Cancer Cell,2003
4. Spontaneous induction of murine pancreatic intraepithelial neoplasia (mPanIN) by acinar cell targeting of oncogenic Kras in adult mice;N Habbe;Proc Natl Acad Sci U S A,2008
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1. Cytokine CCL9 Mediates Oncogenic KRAS-Induced Pancreatic Acinar-to-Ductal Metaplasia by Promoting Reactive Oxygen Species and Metalloproteinases;International Journal of Molecular Sciences;2024-04-26
2. RBPJ Deficiency Sensitizes Pancreatic Acinar Cells to KRAS-Mediated Pancreatic Intraepithelial Neoplasia Initiation;Cellular and Molecular Gastroenterology and Hepatology;2023
3. Notch signaling pathway in pancreatic tumorigenesis;Advances in Cancer Research;2023
4. Inactivation of Notch4 Attenuated Pancreatic Tumorigenesis in Mice;Cancer Research Communications;2022-12-12
5. Generation of Hydrogen Peroxide and Downstream Protein Kinase D1 Signaling Is a Common Feature of Inducers of Pancreatic Acinar-to-Ductal Metaplasia;Antioxidants;2022-01-08
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