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Contact Sensitizers Induce Skin Inflammation via ROS Production and Hyaluronic Acid Degradation

  • Published:2012-07-25 Issue:7 Volume:7 Page:e41340
  • ISSN:1932-6203
  • Container-title:PLoS ONE
  • language:en
  • Short-container-title:PLoS ONE

Author:

Esser Philipp R.,Wölfle Ute,Dürr Christoph,von Loewenich Friederike D.,Schempp Christoph M.,Freudenberg Marina A.,Jakob Thilo,Martin Stefan F.

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference65 articles.

1. T lymphocyte-mediated immune responses to chemical haptens and metal ions: implications for allergic and autoimmune disease.;SF Martin;Int Arch Allergy Immunol,2004

2. Effector and regulatory mechanisms in allergic contact dermatitis.;M Vocanson;Allergy,2009

3. The role of CD4+ and CD8+ T cells in contact hypersensitivity and allergic contact dermatitis.;P Saint-Mezard;Eur J Dermatol,2004

4. Fas-mediated inhibition of CD4+ T cell priming results in dominance of type 1 CD8+ T cells in the immune response to the contact sensitizer trinitrophenyl.;SF Martin;J Immunol,2004

5. Allergic contact dermatitis: the cellular effectors.;I Kimber;Contact Dermatitis,2002

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