CNS SIRT3 Expression Is Altered by Reactive Oxygen Species and in Alzheimer’s Disease-Reference-Cited by-同舟云学术

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CNS SIRT3 Expression Is Altered by Reactive Oxygen Species and in Alzheimer’s Disease

Published:2012-11-06 Issue:11 Volume:7 Page:e48225
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Weir Heather J. M.,Murray Tracey K.,Kehoe Patrick G.,Love Seth,Verdin Eric M.,O’Neill Michael J.,Lane Jon D.,Balthasar Nina

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference25 articles.

1. Loss of perforated synapses in the dentate gyrus: morphological substrate of memory deficit in aged rats;Y Geinisman;Proc Natl Acad Sci U S A,1986

2. Mitochondria are a direct site of A beta accumulation in Alzheimer’s disease neurons: implications for free radical generation and oxidative damage in disease progression;M Manczak;Hum Mol Genet,2006

3. beta-Amyloid peptide induces ultrastructural changes in synaptosomes and potentiates mitochondrial dysfunction in the presence of ryanodine;X Mungarro-Menchaca;J Neurosci Res,2002

4. SIRT3 regulates mitochondrial fatty-acid oxidation by reversible enzyme deacetylation;MD Hirschey;Nature,2010

5. A role for the mitochondrial deacetylase Sirt3 in regulating energy homeostasis;BH Ahn;Proc Natl Acad Sci U S A,2008

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