Targeted Disruption of Ephrin B1 in Cells of Myeloid Lineage Increases Osteoclast Differentiation and Bone Resorption in Mice-Reference-Cited by-同舟云学术

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Targeted Disruption of Ephrin B1 in Cells of Myeloid Lineage Increases Osteoclast Differentiation and Bone Resorption in Mice

Published:2012-03-05 Issue:3 Volume:7 Page:e32887
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Cheng Shaohong,Zhao Shien Lucy,Nelson Brittany,Kesavan Chandrasekhar,Qin Xuezhong,Wergedal Jon,Mohan Subburaman,Xing Weirong

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference46 articles.

1. Inhibition of gap junction communication at ectopic Eph/ephrin boundaries underlies craniofrontonasal syndrome.;A Davy;PLoS Biol,2006

2. Mutations of ephrin-B1 (EFNB1), a marker of tissue boundary formation, cause craniofrontonasal syndrome.;SR Twigg;Proc Natl Acad Sci U S A,2004

3. Target cells in bone for parathormone and calcitonin are different: enrichment for each cell type by sequential digestion of mouse calvaria and selective adhesion to polymeric surfaces.;GL Wong;Proc Natl Acad Sci U S A,1975

4. Eph receptors and ephrins.;PL Jensen;Stem Cells,2000

5. Ephrin-B1 forward and reverse signaling are required during mouse development.;A Davy;Genes Dev,2004

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