Mutation of von Hippel–Lindau Tumour Suppressor and Human Cardiopulmonary Physiology-Reference-Cited by-同舟云学术

Mutation of von Hippel–Lindau Tumour Suppressor and Human Cardiopulmonary Physiology

Published:2006-06-20 Issue:7 Volume:3 Page:e290
ISSN:1549-1676
Container-title:PLoS Medicine
language:en
Short-container-title:PLoS Med

Author:

Smith Thomas G,Brooks Jerome T,Balanos George M,Lappin Terence R,Layton D. Mark,Leedham Dawn L,Liu Chun,Maxwell Patrick H,McMullin Mary F,McNamara Christopher J,Percy Melanie J,Pugh Christopher W,Ratcliffe Peter J,Talbot Nick P,Treacy Marilyn,Robbins Peter A

Publisher

Public Library of Science (PLoS)

Subject

General Medicine

Reference44 articles.

1. Hydroxylation of HIF-1: Oxygen Sensing at the Molecular Level

2. The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis

3. Ubiquitination of hypoxia-inducible factor requires direct binding to the β-domain of the von Hippel–Lindau protein

4. Targeting of HIF-α to the von Hippel-Lindau Ubiquitylation Complex by O 2 -Regulated Prolyl Hydroxylation

5. HIFα Targeted for VHL-Mediated Destruction by Proline Hydroxylation: Implications for O 2 Sensing

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