Insulin Resistance in the Offspring of Parents with Type 2 Diabetes
Author:
Publisher
Public Library of Science (PLoS)
Subject
General Medicine
Reference13 articles.
1. Decreased Insulin-Stimulated ATP Synthesis and Phosphate Transport in Muscle of Insulin-Resistant Offspring of Type 2 Diabetic Parents
2. Impaired Mitochondrial Activity in the Insulin-Resistant Offspring of Patients with Type 2 Diabetes
3. Lipid-Induced Insulin Resistance in Human Muscle Is Associated With Changes in Diacylglycerol, Protein Kinase C, and IκB-α
4. The molecular mechanism linking muscle fat accumulation to insulin resistance
5. Mechanism by Which Fatty Acids Inhibit Insulin Activation of Insulin Receptor Substrate-1 (IRS-1)-associated Phosphatidylinositol 3-Kinase Activity in Muscle
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2. Do static and dynamic insulin resistance indices perform similarly in predicting pre-diabetes and type 2 diabetes?;Diabetes Research and Clinical Practice;2014-08
3. Physical Activity Is the Key Determinant of Skeletal Muscle Mitochondrial Function in Type 2 Diabetes;The Journal of Clinical Endocrinology & Metabolism;2012-09-01
4. Comparison of in vivo postexercise phosphocreatine recovery and resting ATP synthesis flux for the assessment of skeletal muscle mitochondrial function;American Journal of Physiology-Cell Physiology;2010-11
5. Increased mitochondrial content rescuesin vivomuscle oxidative capacity in long‐term high‐fat‐diet‐fed rats;The FASEB Journal;2009-12-29
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