The abundance of the ARL2 GTPase and its GAP, ELMOD2, at mitochondria are modulated by the fusogenic activity of mitofusins and stressors-Reference-Cited by-同舟云学术

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The abundance of the ARL2 GTPase and its GAP, ELMOD2, at mitochondria are modulated by the fusogenic activity of mitofusins and stressors

Published:2017-04-05 Issue:4 Volume:12 Page:e0175164
ISSN:1932-6203
Container-title:PLOS ONE
language:en
Short-container-title:PLoS ONE

Author:

Newman Laura E.,Schiavon Cara R.,Zhou Chengjing,Kahn Richard A.

Funder

National Institute of General Medical Sciences

National Institute of Neurological Disorders and Stroke

American Heart Association

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference66 articles.

1. Mitochondrial Fusion and Fission in Mammals;DC Chan;Annu Rev Cell Dev Biol,2006

2. Dynamics of mitochondrial morphology in healthy cells and during apoptosis;M Karbowski;Cell Death Differ,2003

3. Energetic requirements and bioenergetic modulation of mitochondrial morphology and dynamics;C Sauvanet;Semin Cell Dev Biol,2010

4. C. elegans dynamin-related protein DRP-1 controls severing of the mitochondrial outer membrane;AM Labrousse;Mol Cell,1999

5. Dynamin-related protein Drp1 is required for mitochondrial division in mammalian cells;E Smirnova;Mol Biol Cell,2001

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3. The Arf family GTPases: Regulation of vesicle biogenesis and beyond;BioEssays;2023-03-30

4. Mitofusins: from mitochondria to fertility;Cellular and Molecular Life Sciences;2022-06-20

5. The ARF GAPs ELMOD1 and ELMOD3 act at the Golgi and cilia to regulate ciliogenesis and ciliary protein traffic;Molecular Biology of the Cell;2022-02-01

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