DNA Damage Stress and Inhibition of Jak2-V617F Cause Its Degradation and Synergistically Induce Apoptosis through Activation of GSK3β-Reference-Cited by-同舟云学术

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DNA Damage Stress and Inhibition of Jak2-V617F Cause Its Degradation and Synergistically Induce Apoptosis through Activation of GSK3β

Published:2011-11-08 Issue:11 Volume:6 Page:e27397
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Nagao Toshikage,Oshikawa Gaku,Wu Nan,Kurosu Tetsuya,Miura Osamu

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Cited by 14 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Silibinin eliminates mitochondrial ROS and restores autophagy through IL6ST/JAK2/STAT3 signaling pathway to protect cardiomyocytes from doxorubicin-induced injury;European Journal of Pharmacology;2022-08

2. Role of CBL Mutations in Cancer and Non-Malignant Phenotype;Cancers;2022-02-08

3. Negative Regulation of the Innate Immune Response through Proteasomal Degradation and Deubiquitination;Viruses;2021-03-30

4. Inhibition of USP9X Downregulates JAK2-V617F and Induces Apoptosis Synergistically with BH3 Mimetics Preferentially in Ruxolitinib-Persistent JAK2-V617F-Positive Leukemic Cells;Cancers;2020-02-10

5. Ameliorating oxidative stress and inflammation by Hesperidin and vitamin E in doxorubicin induced cardiomyopathy;Turkish Journal of Biochemistry;2018-09-05

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