PKC-Dependent GlyT1 Ubiquitination Occurs Independent of Phosphorylation: Inespecificity in Lysine Selection for Ubiquitination-Reference-Cited by-同舟云学术

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PKC-Dependent GlyT1 Ubiquitination Occurs Independent of Phosphorylation: Inespecificity in Lysine Selection for Ubiquitination

Published:2015-09-29 Issue:9 Volume:10 Page:e0138897
ISSN:1932-6203
Container-title:PLOS ONE
language:en
Short-container-title:PLoS ONE

Author:

Barrera Susana P.,Castrejon-Tellez Vicente,Trinidad Margarita,Robles-Escajeda Elisa,Vargas-Medrano Javier,Varela-Ramirez Armando,Miranda Manuel

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference55 articles.

1. Glycine potentiates the NMDA response in cultured mouse brain neurons;JW Johnson;Nature,1987

2. Sustained saturating level of glycine induces changes in NR2B-containing-NMDA receptor localization in the CA1 region of the hippocampus;Y Imamura;Journal of Neurochemistry,2008

3. Modulation of N-methyl-D-aspartate receptor function by glycine transport;R Bergeron;PNAS,1998

4. Control of NMDA receptor activation by a glycine transporter co-expressed in Xenopus oocytes;S Supplisson;J Neurosci,1997

5. Localization of the GLYT1 Glycine Transporter at Glutamatergic Synapses in the Rat Brain;B Cubelos;Cereb Cortex,2005

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