Testisin/Prss21 deficiency causes increased vascular permeability and a hemorrhagic phenotype during luteal angiogenesis-Reference-Cited by-同舟云学术

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Testisin/Prss21 deficiency causes increased vascular permeability and a hemorrhagic phenotype during luteal angiogenesis

Published:2020-06-08 Issue:6 Volume:15 Page:e0234407
ISSN:1932-6203
Container-title:PLOS ONE
language:en
Short-container-title:PLoS ONE

Author:

Peroutka Raymond J.,Buzza Marguerite S.,Mukhopadhyay Subhradip,Johnson Tierra A.,Driesbaugh Kathryn H.,Antalis Toni M.^ORCID

Funder

National Cancer Institute

National Heart, Lung, and Blood Institute

Biomedical Laboratory Research and Development, VA Office of Research and Development

American Cancer Society

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference54 articles.

1. Molecular regulation of angiogenesis and lymphangiogenesis;RH Adams;Nat Rev Mol Cell Biol,2007

2. Recent molecular discoveries in angiogenesis and antiangiogenic therapies in cancer;J Welti;J Clin Invest,2013

3. Transforming growth factor-beta: vasculogenesis, angiogenesis, and vessel wall integrity;MS Pepper;Cytokine Growth Factor Rev,1997

4. A unifying concept in vascular health and disease;MA Schwartz;Science,2018

5. Disorders of Vascular Permeability;C Park-Windhol;Annu Rev Pathol,2016

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1. scRNA-Seq and Bulk-Seq Analysis Identifies S100A9 Plasma Cells as a Potentially Effective Immunotherapeutic Agent for Multiple Myeloma;Journal of Inflammation Research;2024-03

2. Intersection of Coagulation and Fibrinolysis by the Glycosylphosphatidylinositol (GPI)-Anchored Serine Protease Testisin;International Journal of Molecular Sciences;2023-05-26

3. Extracellular: Plasma Membrane Proteases – Serine Proteases;Encyclopedia of Cell Biology;2023

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