Mycobacterium tuberculosis WhiB3 Maintains Redox Homeostasis by Regulating Virulence Lipid Anabolism to Modulate Macrophage Response-Reference-Cited by-同舟云学术

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Mycobacterium tuberculosis WhiB3 Maintains Redox Homeostasis by Regulating Virulence Lipid Anabolism to Modulate Macrophage Response

Published:2009-08-14 Issue:8 Volume:5 Page:e1000545
ISSN:1553-7374
Container-title:PLoS Pathogens
language:en
Short-container-title:PLoS Pathog

Author:

Singh Amit,Crossman David K.,Mai Deborah,Guidry Loni,Voskuil Martin I.,Renfrow Matthew B.,Steyn Adrie J. C.

Publisher

Public Library of Science (PLoS)

Subject

Virology,Genetics,Molecular Biology,Immunology,Microbiology,Parasitology

Reference48 articles.

1. Global Burden of Tuberculosis: Estimated Incidence, Prevalence, and Mortality by Country.;C Dye;JAMA,1999

2. Microbial glycolipids: possible virulence factors that scavenge oxygen radicals.;J Chan;Proc Natl Acad Sci U S A,1989

3. A glycolipid of hypervirulent tuberculosis strains that inhibits the innate immune response.;MB Reed;Nature,2004

4. Disparate responses to oxidative stress in saprophytic and pathogenic mycobacteria.;DR Sherman;Proc Natl Acad Sci U S A,1995

5. Analysis of the phthiocerol dimycocerosate locus of Mycobacterium tuberculosis. Evidence that this lipid is involved in the cell wall permeability barrier.;LR Camacho;J Biol Chem,2001

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