Host cell depletion of tryptophan by IFNγ-induced Indoleamine 2,3-dioxygenase 1 (IDO1) inhibits lysosomal replication of Coxiella burnetii

Author:

Ganesan SandhyaORCID,Roy Craig R.ORCID

Funder

National Institute of Allergy and Infectious Diseases

Publisher

Public Library of Science (PLoS)

Subject

Virology,Genetics,Molecular Biology,Immunology,Microbiology,Parasitology

Reference64 articles.

1. Q fever;M Maurin;Clin Microbiol Rev,1999

2. Effector protein translocation by the Coxiella burnetii Dot/Icm type IV secretion system requires endocytic maturation of the pathogen-occupied vacuole;HJ Newton;PLoS ONE. Public Library of Science,2013

3. The Coxiella burnetii Dot/Icm system delivers a unique repertoire of type IV effectors into host cells and is required for intracellular replication;KL Carey;PLoS Pathogens. Public Library of Science,2011

4. Biogenesis of the lysosome-derived vacuole containing Coxiella burnetii;LJ Kohler;Microbes Infect,2015

5. Dot/Icm type IVB secretion system requirements for Coxiella burnetii growth in human macrophages;PA Beare;mBio. American Society for Microbiology,2011

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