The Human Papillomavirus E6 Oncogene Represses a Cell Adhesion Pathway and Disrupts Focal Adhesion through Degradation of TAp63β upon Transformation-Reference-Cited by-同舟云学术

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The Human Papillomavirus E6 Oncogene Represses a Cell Adhesion Pathway and Disrupts Focal Adhesion through Degradation of TAp63β upon Transformation

Published:2011-09-29 Issue:9 Volume:7 Page:e1002256
ISSN:1553-7374
Container-title:PLoS Pathogens
language:en
Short-container-title:PLoS Pathog

Author:

Ben Khalifa Youcef,Teissier Sébastien,Tan Meng-Kwang Marcus,Phan Quang Tien,Daynac Mathieu,Wong Wei Qi,Thierry Françoise

Publisher

Public Library of Science (PLoS)

Subject

Virology,Genetics,Molecular Biology,Immunology,Microbiology,Parasitology

Reference50 articles.

1. Human papillomavirus is a necessary cause of invasive cervical cancer worldwide.;JM Walboomers;J Pathol,1999

2. The majority of viral-cellular fusion transcripts in cervical carcinomas cotranscribe cellular sequences of known or predicted genes.;I Kraus;Cancer Res,2008

3. Type-dependent integration frequency of human papillomavirus genomes in cervical lesions.;S Vinokurova;Cancer Res,2008

4. A genomic approach reveals a novel mitotic pathway in papillomavirus carcinogenesis.;F Thierry;Cancer Res,2004

5. A new E6/P63 pathway, together with a strong E7/E2F mitotic pathway, modulates the transcriptome in cervical cancer cells.;S Teissier;J Virol,2007

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