Influenza virus differentially activates mTORC1 and mTORC2 signaling to maximize late stage replication-Reference-Cited by-同舟云学术

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Influenza virus differentially activates mTORC1 and mTORC2 signaling to maximize late stage replication

Published:2017-09-27 Issue:9 Volume:13 Page:e1006635
ISSN:1553-7374
Container-title:PLOS Pathogens
language:en
Short-container-title:PLoS Pathog

Author:

Kuss-Duerkop Sharon K.^ORCID,Wang Juan,Mena Ignacio^ORCID,White Kris^ORCID,Metreveli Giorgi,Sakthivel Ramanavelan,Mata Miguel A.,Muñoz-Moreno Raquel,Chen Xiang,Krammer Florian^ORCID,Diamond Michael S.,Chen Zhijian J.,García-Sastre Adolfo^ORCID,Fontoura Beatriz M. A.^ORCID

Funder

National Institute of General Medical Sciences

Cancer Prevention and Research Institute of Texas

National Institute of Allergy and Infectious Diseases

Publisher

Public Library of Science (PLoS)

Subject

Virology,Genetics,Molecular Biology,Immunology,Microbiology,Parasitology

Reference89 articles.

1. Influenza virus-host interactome screen as a platform for antiviral drug development;T Watanabe;Cell Host Microbe,2014

2. Meta- and Orthogonal Integration of Influenza "OMICs" Data Defines a Role for UBR4 in Virus Budding;S Tripathi;Cell Host Microbe,2015

3. Cellular networks involved in the influenza virus life cycle;T Watanabe;Cell Host Microbe,2010

4. Human host factors required for influenza virus replication;R König;Nature,2010

5. Chemical inhibition of RNA viruses reveals REDD1 as a host defense factor;MA Mata;Nat Chem Biol,2011

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