Propagation of RML Prions in Mice Expressing PrP Devoid of GPI Anchor Leads to Formation of a Novel, Stable Prion Strain-Reference-Cited by-同舟云学术

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Propagation of RML Prions in Mice Expressing PrP Devoid of GPI Anchor Leads to Formation of a Novel, Stable Prion Strain

Published:2012-06-07 Issue:6 Volume:8 Page:e1002746
ISSN:1553-7374
Container-title:PLoS Pathogens
language:en
Short-container-title:PLoS Pathog

Author:

Mahal Sukhvir Paul,Jablonski Joseph,Suponitsky-Kroyter Irena,Oelschlegel Anja Maria,Herva Maria Eugenia,Oldstone Michael,Weissmann Charles

Publisher

Public Library of Science (PLoS)

Subject

Virology,Genetics,Molecular Biology,Immunology,Microbiology,Parasitology

Reference84 articles.

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2. Glycosylation differences between the normal and pathogenic prion protein isoforms.;PM Rudd;Proc Natl Acad Sci U S A,1999

3. Prion glycoprotein: structure, dynamics, and roles for the sugars.;PM Rudd;Biochemistry,2001

4. Regional heterogeneity of cellular prion protein isoforms in the mouse brain.;V Beringue;Brain,2003

5. Transmissible spongiform encephalopathy strain, PrP genotype and brain region all affect the degree of glycosylation of PrPSc.;RA Somerville;J Gen Virol,2005

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