De Novo Herpes Simplex Virus VP16 Expression Gates a Dynamic Programmatic Transition and Sets the Latent/Lytic Balance during Acute Infection in Trigeminal Ganglia-Reference-Cited by-同舟云学术

De Novo Herpes Simplex Virus VP16 Expression Gates a Dynamic Programmatic Transition and Sets the Latent/Lytic Balance during Acute Infection in Trigeminal Ganglia

Published:2016-09-08 Issue:9 Volume:12 Page:e1005877
ISSN:1553-7374
Container-title:PLOS Pathogens
language:en
Short-container-title:PLoS Pathog

Author:

Sawtell Nancy M.,Thompson Richard L.

Funder

National Institute of Allergy and Infectious Diseases

Publisher

Public Library of Science (PLoS)

Subject

Virology,Genetics,Molecular Biology,Immunology,Microbiology,Parasitology

Reference98 articles.

1. Weller SK (2011) Alphaherpesviruses Molecular Virology. 480 p.

2. Identification of the latency-associated transcript promoter by expression of rabbit beta-globin mRNA in mouse sensory nerve ganglia latently infected with a recombinant herpes simplex virus;AT Dobson;J Virol,1989

3. Pathways of viral gene expression during acute neuronal infection with HSV-1;TP Margolis;Virology,1992

4. Rapid in vivo reactivation of herpes simplex virus in latently infected murine ganglionic neurons after transient hyperthermia;NM Sawtell;J Virol,1992

5. Utilization of the herpes simplex virus type 1 latency-associated regulatory region to drive stable reporter gene expression in the nervous system;RH Lachmann;J Virol,1997

Cited by 41 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Neuronal expression of herpes simplex virus-1 VP16 protein induces pseudorabies virus escape from silencing and reactivation;Journal of Virology;2024-07-23

2. Co-option of mitochondrial nucleic acid sensing pathways by HSV-1 UL12.5 for reactivation from latent Infection;2024-07-07

3. c-Jun signaling during initial HSV-1 infection modulates latency to enhance later reactivation in addition to directly promoting the progression to full reactivation;Journal of Virology;2024-02-20

4. c-Jun Signaling During Initial HSV-1 Infection Modulates Latency to Enhance Later Reactivation in addition to Directly Promoting the Progression to Full Reactivation;2023-11-10

5. Intermittent therapy with helicase-primase inhibitor IM-250 efficiently controls recurrent herpes disease and reduces reactivation of latent HSV;Antiviral Research;2023-11