Impaired Recruitment of Grk6 and β-Arrestin2 Causes Delayed Internalization and Desensitization of a WHIM Syndrome-Associated CXCR4 Mutant Receptor-Reference-Cited by-同舟云学术

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Impaired Recruitment of Grk6 and β-Arrestin2 Causes Delayed Internalization and Desensitization of a WHIM Syndrome-Associated CXCR4 Mutant Receptor

Published:2009-12-01 Issue:12 Volume:4 Page:e8102
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

McCormick Peter J.,Segarra Marta,Gasperini Paola,Gulino A. Virginia,Tosato Giovanna

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Cited by 57 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The complex nature of CXCR4 mutations in WHIM syndrome;Frontiers in Immunology;2024-07-05

2. Reduced G protein signaling despite impaired internalization and β-arrestin recruitment in patients carrying a CXCR4Leu317fsX3 mutation causing WHIM syndrome;JCI Insight;2023-03-08

3. The Role of G Protein-Coupled Receptor Kinase 6 Regulation in Inflammation and Pain;International Journal of Molecular Sciences;2022-12-14

4. Characterization of a new WHIM syndrome mutant reveals mechanistic differences in regulation of the chemokine receptor CXCR4;Journal of Biological Chemistry;2022-02

5. Design, Synthesis, and Characterization of 4-Aminoquinazolines as Potent Inhibitors of the G Protein-Coupled Receptor Kinase 6 (GRK6) for the Treatment of Multiple Myeloma;Journal of Medicinal Chemistry;2021-07-22

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