Hypoxia-inducible factor 1α inhibitor induces cell death via suppression of BCR-ABL1 and Met expression in BCR-ABL1 tyrosine kinase inhibitor sensitive and resistant chronic myeloid leukemia cells
Author:
Affiliation:
1. Division of Pharmacotherapy, Kindai University Faculty of Pharmacy, Kowakae 577-8502, Higashi-Osaka, Japan
2. Department of Pharmacy, Japanese Red Cross Society Wakayama Medical Center, Wakayama 640-8558, Japan
Funder
Japan Society for the Promotion of Science
Publisher
Korean Society for Biochemistry and Molecular Biology - BMB Reports
Subject
Molecular Biology,Biochemistry,General Medicine
Link
http://pdf.medrang.co.kr/BMB/2023/056/BMB056-02-05.pdf
Reference22 articles.
1. Targeting BCR-Abl in the treatment of Philadelphia-chromosome positive chronic myelogenous leukemia
2. JAK/STAT, Raf/MEK/ERK, PI3K/Akt and BCR-ABL in cell cycle progression and leukemogenesis
3. European LeukemiaNet 2020 recommendations for treating chronic myeloid leukemia
4. Current Treatment Options for Chronic Myeloid Leukemia Patients Failing Second-Generation Tyrosine Kinase Inhibitors
5. Resistance to Tyrosine Kinase Inhibitors in Chronic Myeloid Leukemia—From Molecular Mechanisms to Clinical Relevance
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1. Chronic myeloid leukaemia: Biology and therapy;Blood Reviews;2024-05
2. HIF-1α signaling: Essential roles in tumorigenesis and implications in targeted therapies;Genes & Diseases;2024-01
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