BLM Deficiency Is Not Associated with Sensitivity to Hydroxyurea-Induced Replication Stress
Author:
Affiliation:
1. Institut Curie, Centre de Recherche, Centre Universitaire, Bât. 110, 91405 Orsay, France
2. CNRS, UMR 3348, 91405 Orsay, France
Abstract
Funder
Institut Curie (PIC)
Publisher
Hindawi Limited
Subject
Molecular Biology,Biochemistry
Link
http://downloads.hindawi.com/journals/jna/2010/319754.pdf
Reference42 articles.
1. The Bloom's syndrome gene product is homologous to RecQ helicases
2. Functional interactions between BLM and XRCC3 in the cell
3. Sister Chromatid Exchanges Are Mediated by Homologous Recombination in Vertebrate Cells
4. The Bloom Syndrome Protein Limits the Lethality Associated with RAD51 Deficiency
5. A retarded rate of DNA chain growth in Bloom's syndrome.
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1. A POLD3/BLM dependent pathway handles DSBs in transcribed chromatin upon excessive RNA:DNA hybrid accumulation;Nature Communications;2022-04-19
2. The Bloom syndrome complex senses RPA-coated single-stranded DNA to restart stalled replication forks;Nature Communications;2021-01-26
3. Differential contribution of the Fanconi anemia-related proteins to repair of several types of DNA damage in cultured silkworm cells;FEBS Letters;2014-09-19
4. Distinct functions of human RECQ helicases WRN and BLM in replication fork recovery and progression after hydroxyurea-induced stalling;DNA Repair;2013-02
5. The Werner and Bloom Syndrome Proteins Help Resolve Replication Blockage by Converting (Regressed) Holliday Junctions to Functional Replication Forks;Biochemistry;2011-07-21
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