Polymorphisms of Genes Associated with Intracellular Signaling Pathways in Juvenile Idiopathic Arthritis
Author:
Publisher
Allerton Press
Subject
Genetics,Agricultural and Biological Sciences (miscellaneous),Cell Biology
Link
https://link.springer.com/content/pdf/10.3103/S0095452722030070.pdf
Reference26 articles.
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2. Banerjee, S., Biehl, A., Gadina, M., et al., JAK–STAT Signaling as a target for inflammatory and autoimmune diseases: Current and future prospects, Drugs, 2017, vol. 77, pp. 521–546. https://doi.org/10.1007/s40265-017-0701-9
3. Baralle, D. and Baralle, M., Splicing in action: assessing disease causing sequence changes, J. Med. Genet., 2005, vol. 42, no. 10, pp. 737–748. PMID: 16199547; PMCID: PMC1735933.https://doi.org/10.1136/jmg.2004.029538
4. Buratti, E., Chivers, M., Královicová, J., et al., Aber-rant 5′ splice sites in human disease genes: mutation pattern, nucleotide structure and comparison of computational tools that predict their utilization, Nucleic Acids Res., 2007, vol. 35, no. 13, pp. 4250–4263. https://doi.org/10.1093/nar/gkm402
5. Ciccarelli, F., De Martinis, M., and Ginaldi, L., An update on autoinflammatory diseases, Curr. Med. Chem., 2014, vol. 21, no. 3, pp. 261–269.
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