Response to comment by Moxon et al.
Author:
Affiliation:
1. Institute of medical sciences, Aberdeen Cardiovascular and Diabetes Centre, School of Medicine, Medical Sciences and Nutrition, University of Aberdeen, Aberdeen, U.K.
Publisher
Portland Press Ltd.
Subject
General Medicine
Link
http://portlandpress.com/clinsci/article-pdf/132/1/39/450576/cs-2017-1555.pdf
Reference8 articles.
1. Comment on ‘Pharmacological inhibition of protein tyrosine phosphatase 1B protects against atherosclerotic plaque formation in the LDLR-/- mouse model of atherosclerosis’;Moxon;Clin. Sci. (Lond.),2018
2. Pharmacological inhibition of protein tyrosine phosphatase 1B protects against atherosclerotic plaque formation in the LDLR-/- mouse model of atherosclerosis;Thompson;Clin. Sci. (Lond.),2017
3. Myeloid protein tyrosine phosphatase 1B (PTP1B) deficiency protects against atherosclerotic plaque formation in the ApoE-/- mouse model of atherosclerosis with alterations in IL10/AMPKα pathway;Thompson;Mol. Metab.,2017
4. Apparent receptor-mediated activation of Ca2+-dependent conductive Cl- transport by shark-derived polyaminosterols;Chernova;Am. J. Physiol. Regul. Integr. Comp. Physiol.,2005
5. Appetite suppression and weight reduction by a centrally active aminosterol;Ahima;Diabetes,2002
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