PTP1B Suppresses Prolactin Activation of Stat5 in Breast Cancer Cells-Reference-Cited by-同舟云学术

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PTP1B Suppresses Prolactin Activation of Stat5 in Breast Cancer Cells

Published:2010-12 Issue:6 Volume:177 Page:2971-2983
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Johnson Kevin J.,Peck Amy R.,Liu Chengbao,Tran Thai H.,Utama Fransiscus E.,Sjolund Ashley B.,Schaber John D.,Witkiewicz Agnieszka K.,Rui Hallgeir

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference71 articles.

1. Stat5a and Stat5b proteins have essential and nonessential, or redundant, roles in cytokine responses;Teglund;Cell,1998

2. Basal activation of transcription factor signal transducer and activator of transcription (Stat5) in nonpregnant mouse and human breast epithelium;Nevalainen;Mol Endocrinol,2002

3. Stat5a is mandatory for adult mammary gland development and lactogenesis;Liu;Genes Dev,1997

4. Signal transducer and activator of transcription (Stat) 5 controls the proliferation and differentiation of mammary alveolar epithelium;Miyoshi;J Cell Biol,2001

5. Inactivation of Stat5 in mouse mammary epithelium during pregnancy reveals distinct functions in cell proliferation, survival, and differentiation;Cui;Mol Cell Biol,2004

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1. Protein Tyrosine Phosphatase 1B (PTP1B): A Comprehensive Review of Its Role in Pathogenesis of Human Diseases;International Journal of Molecular Sciences;2024-06-27

2. Reduction of phosphorylated signal transducer and activator of transcription-5 expression in feline mammary carcinoma;Journal of Veterinary Medical Science;2024

3. STAT family of transcription factors in breast cancer: Pathogenesis and therapeutic opportunities and challenges;Seminars in Cancer Biology;2022-11

4. Prolactin receptor gene transcriptional control, regulatory modalities relevant to breast cancer resistance and invasiveness;Frontiers in Endocrinology;2022-09-15

5. Protein Tyrosine Phosphatase 1B (PTP1B): Insights into its New Implications in Tumorigenesis;Current Cancer Drug Targets;2022-03

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