Caveolin-1 (P132L), a Common Breast Cancer Mutation, Confers Mammary Cell Invasiveness and Defines a Novel Stem Cell/Metastasis-Associated Gene Signature-Reference-Cited by-同舟云学术

Caveolin-1 (P132L), a Common Breast Cancer Mutation, Confers Mammary Cell Invasiveness and Defines a Novel Stem Cell/Metastasis-Associated Gene Signature

Published:2009-05 Issue:5 Volume:174 Page:1650-1662
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Bonuccelli Gloria,Casimiro Mathew C.,Sotgia Federica,Wang Chenguang,Liu Manran,Katiyar Sanjay,Zhou Jie,Dew Elliott,Capozza Franco,Daumer Kristin M.,Minetti Carlo,Milliman Janet N.,Alpy Fabien,Rio Marie-Christine,Tomasetto Catherine,Mercier Isabelle,Flomenberg Neal,Frank Philippe G.,Pestell Richard G.,Lisanti Michael P.

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference86 articles.

1. Invasion activating caveolin-1 mutation in human scirrhous breast cancers;Hayashi;Cancer Res,2001

2. Caveolin-1 mutations in human breast cancer: functional association with estrogen receptor alpha-positive status;Li;Am J Pathol,2006

3. Caveolin-1 mutations (P132L and null) and the pathogenesis of breast cancer: caveolin-1 (P132L) behaves in a dominant-negative manner and caveolin-1 (−/−) null mice show mammary epithelial cell hyperplasia;Lee;Am J Pathol,2002

4. Caveolin-1-deficient mice show accelerated mammary gland development during pregnancy, premature lactation, and hyperactivation of the Jak-2/STAT5a signaling cascade;Park;Mol Biol Cell,2002

5. Loss of caveolin-1 gene expression accelerates the development of dysplastic mammary lesions in tumor-prone transgenic mice;Williams;Mol Biol Cell,2003

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