Keratinocyte but Not Endothelial Cell-Specific Overexpression of Tie2 Leads to the Development of Psoriasis-Reference-Cited by-同舟云学术

Keratinocyte but Not Endothelial Cell-Specific Overexpression of Tie2 Leads to the Development of Psoriasis

Published:2009-04 Issue:4 Volume:174 Page:1443-1458
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Wolfram Julie A.,Diaconu Doina,Hatala Denise A.,Rastegar Jessica,Knutsen Dorothy A.,Lowther Abigail,Askew David,Gilliam Anita C.,McCormick Thomas S.,Ward Nicole L.

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference55 articles.

1. Mouse models of psoriasis;Gudjonsson;J Invest Dermatol,2007

2. Animal models of psoriasis and psoriatic arthritis: an update;Conrad;Curr Rheumatol Rep,2006

3. Transgenic delivery of VEGF to mouse skin leads to an inflammatory condition resembling human psoriasis;Xia;Blood,2003

4. Induction of cutaneous delayed-type hypersensitivity reactions in VEGF-A transgenic mice results in chronic skin inflammation associated with persistent lymphatic hyperplasia;Kunstfeld;Blood,2004

5. A cyclosporine-sensitive psoriasis-like disease produced in Tie2 transgenic mice;Voskas;Am J Pathol,2005

Cited by 76 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Macrophage Functions in Psoriasis: Lessons from Mouse Models;International Journal of Molecular Sciences;2024-05-13

2. RNA-Based Antipsoriatic Gene Therapy: An Updated Review Focusing on Evidence from Animal Models;Drug Design, Development and Therapy;2024-04

3. Casting for Proteins in Psoriatic Inflammation Hooks Glycyl-tRNA Synthetases (GARS);Journal of Investigative Dermatology;2024-04

4. Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling;JCI Insight;2024-03-12

5. Evaluation of Kynu, Defb2, Camp, and Penk Expression Levels as Psoriasis Marker in the Imiquimod‐Induced Psoriasis Model;Mediators of Inflammation;2024-01