Defective Mitochondrial Adenosine Triphosphate Production in Skeletal Muscle From Patients With Dominant Optic Atrophy Due to OPA1 Mutations-Reference-Cited by-同舟云学术

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Defective Mitochondrial Adenosine Triphosphate Production in Skeletal Muscle From Patients With Dominant Optic Atrophy Due to OPA1 Mutations

Published:2011-01-01 Issue:1 Volume:68 Page:
ISSN:0003-9942
Container-title:Archives of Neurology
language:en
Short-container-title:Arch Neurol

Author:

Lodi Raffaele,Tonon Caterina,Valentino Maria Lucia,Manners David,Testa Claudia,Malucelli Emil,La Morgia Chiara,Barboni Piero,Carbonelli Michele,Schimpf Simone,Wissinger Bernd,Zeviani Massimo,Baruzzi Agostino,Liguori Rocco,Barbiroli Bruno,Carelli Valerio

Publisher

American Medical Association (AMA)

Subject

Clinical Neurology,Arts and Humanities (miscellaneous)

Cited by 41 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Creation of an Isogenic Human iPSC-Based RGC Model of Dominant Optic Atrophy Harboring the Pathogenic Variant c.1861C>T (p.Gln621Ter) in the OPA1 Gene;International Journal of Molecular Sciences;2024-06-30

2. Circadian timing of satellite cell function and muscle regeneration;Current Topics in Developmental Biology;2024

3. Opa1 and Drp1 reciprocally regulate cristae morphology, ETC function, and NAD+ regeneration in KRas-mutant lung adenocarcinoma;Cell Reports;2022-12

4. Opa1 and Drp1 reciprocally regulate cristae morphology, ETC function, and NAD⁺ regeneration in KRas-mutant lung adenocarcinoma;2022-06-01

5. Therapeutic applications of mitochondrial transplantation;Biochimie;2022-04

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