Assessment of Clinical Response to Janus Kinase Inhibition in Patients With Familial Chilblain Lupus andTREX1Mutation-Reference-Cited by-同舟云学术

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Assessment of Clinical Response to Janus Kinase Inhibition in Patients With Familial Chilblain Lupus andTREX1Mutation

Published:2019-03-01 Issue:3 Volume:155 Page:342
ISSN:2168-6068
Container-title:JAMA Dermatology
language:en
Short-container-title:JAMA Dermatol

Author:

Zimmermann Nick¹,Wolf Christine²,Schwenke Reiner³,Lüth Anne¹,Schmidt Franziska¹,Engel Kerstin²,Lee-Kirsch Min Ae²,Günther Claudia¹

Affiliation:

1. Department of Dermatology, Medical Faculty Carl Gustav Carus, Technical University Dresden, Dresden, Germany

2. Department of Pediatrics, Medical Faculty Carl Gustav Carus, Technical University Dresden, Dresden, Germany

3. Rheumatological Practice, Dresden, Germany

Publisher

American Medical Association (AMA)

Subject

Dermatology

Link

http:///jamadermatology/article-pdf/155/3/342/17393058/jamadermatology_zimmermann_2019_br_180039.pdf

Reference17 articles.

1. RPA and Rad51 constitute a cell intrinsic mechanism to protect the cytosol from self DNA.;Wolf;Nat Commun,2016

2. The type I interferonopathies.;Lee-Kirsch;Annu Rev Med,2017

3. Heterozygous mutations in TREX1 cause familial chilblain lupus and dominant Aicardi-Goutieres syndrome.;Rice;Am J Hum Genet,2007

4. Deregulated type I IFN response in TREX1-associated familial chilblain lupus.;Peschke;J Invest Dermatol,2014

5. The cGAS-cGAMP-STING pathway connects DNA damage to inflammation, senescence, and cancer.;Li;J Exp Med,2018

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