Decrease of Anti-DSG3, but Not Anti-DSG1 Antibody, After Cessation of Sitagliptin Treatment in a Patient With Pemphigus Vulgaris
Author:
Affiliation:
1. Department of Dermatology, Sapporo Medical University, Sapporo, Japan
Publisher
American Medical Association (AMA)
Subject
Dermatology
Link
http:///jamadermatology/article-pdf/155/3/391/17393123/jamadermatology_handa_2019_lo_180039.pdf
Reference5 articles.
1. Pemphigus vulgaris developing after 6-month treatment with a dipeptidyl peptidase-4 inhibitor: a case report.;Okauchi;J Dermatol,2018
2. Clinical and immunological profiles in 17 Japanese patients with drug-induced pemphigus studied at Kurume University.;Yoshimura;Br J Dermatol,2014
3. HLA-DQB1*03:01 as a biomarker for genetic susceptibility to bullous pemphigoid induced by DPP-4 inhibitors.;Ujiie;J Invest Dermatol,2018
4. A common major histocompatibility complex class II allele HLA-DQB1*0301 is present in clinical variants of pemphigoid.;Delgado;Proc Natl Acad Sci U S A,1996
5. HLA class II (DRB, DQA1 and DQB1) allele and haplotype frequencies in the patients with pemphigus vulgaris.;Shams;J Clin Immunol,2009
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1. Dipeptidyl peptidase‐4 inhibitor‐related pemphigus herpetiformis with antibodies against desmocollin 3;The Journal of Dermatology;2023-09-07
2. 36-month follow-up of a pure sensory mononeuritis multiplex and IgG1 deficiency improved after treatment with sitagliptin and Vitamin D3;EUR REV MED PHARMACO;2021
3. Prednisolone/sitagliptin;Reactions Weekly;2019-04
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