<i>APOE</i> Genotype and White Matter Hyperintensities in Sporadic Alzheimer Disease-Reference-Cited by-同舟云学术

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APOE Genotype and White Matter Hyperintensities in Sporadic Alzheimer Disease

Published:2024-06-01 Issue:6 Volume:81 Page:662
ISSN:2168-6149
Container-title:JAMA Neurology
language:en
Short-container-title:JAMA Neurol

Author:

Mirza Saira¹,Wilkinson Tim²,Masellis Mario¹³

Affiliation:

1. L. C. Campbell Cognitive Neurology Research Unit, Hurvitz Brain Sciences Program, Sunnybrook Research Institute, Toronto, Ontario, Canada

2. Centre for Clinical Brain Sciences, University of Edinburgh, Edinburgh, United Kingdom

3. Division of Neurology, Department of Medicine, Sunnybrook Health Sciences Centre and the University of Toronto, Toronto, Ontario, Canada

Publisher

American Medical Association (AMA)

Link

https://jamanetwork.com/journals/jamaneurology/articlepdf/2817633/jamaneurology_mirza_2024_le_240003_1717440915.04403.pdf

Reference5 articles.

1. Etiology of white matter hyperintensities in autosomal dominant and sporadic Alzheimer disease.;Shirzadi;JAMA Neurol,2023

2. APOE e4, white matter hyperintensities, and cognition in Alzheimer and Lewy body dementia.;Mirza;Neurology,2019

3. APOE associations with severe CAA-associated vasculopathic changes: collaborative meta-analysis.;Rannikmäe;J Neurol Neurosurg Psychiatry,2014

4. Patterns of atrophy in pathologically confirmed dementias: a voxelwise analysis.;Harper;J Neurol Neurosurg Psychiatry,2017

5. The cortical signature of Alzheimer’s disease: regionally specific cortical thinning relates to symptom severity in very mild to mild AD dementia and is detectable in asymptomatic amyloid-positive individuals.;Dickerson;Cereb Cortex,2009

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