Apolipoprotein E Type 4 Allele and Cerebral Glucose Metabolism in Relatives at Risk for Familial Alzheimer Disease
Author:
Publisher
American Medical Association (AMA)
Subject
General Medicine
Cited by 326 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Effects of APOE e4 and Neuropathological Diagnoses on Neuropsychiatric Symptoms: Mediation Analyses and Likely Causation in an Integrated NACC Database;2024-01-30
2. Increased glucose metabolism and impaired glutamate transport in human astrocytes are potential early triggers of abnormal extracellular glutamate accumulation in hiPSC‐derived models of Alzheimer's disease;Journal of Neurochemistry;2023-12-08
3. Alzheimer's disease phenotype based upon the carrier status of the apolipoprotein E ɛ4 allele;Brain Pathology;2023-08-30
4. Hypometabolism, Alzheimer’s Disease, and Possible Therapeutic Targets: An Overview;Cells;2023-08-08
5. The Novel Role of Mitochondrial Citrate Synthase and Citrate in the Pathophysiology of Alzheimer’s Disease;Journal of Alzheimer's Disease;2023-07-25
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