A Chimeric Cfh Transgene Leads to Increased Retinal Oxidative Stress, Inflammation, and Accumulation of Activated Subretinal Microglia in Mice-Reference-Cited by-同舟云学术

A Chimeric Cfh Transgene Leads to Increased Retinal Oxidative Stress, Inflammation, and Accumulation of Activated Subretinal Microglia in Mice

Published:2015-06-01 Issue:6 Volume:56 Page:3427
ISSN:1552-5783
Container-title:Investigative Opthalmology & Visual Science
language:en
Short-container-title:Invest. Ophthalmol. Vis. Sci.

Author:

Aredo Bogale¹,Li Tao²,Chen Xiao¹,Zhang Kaiyan¹,Wang Cynthia Xin-Zhao¹,Gou Darlene¹,Zhao Biren¹,He Yuguang¹,Ufret-Vincenty Rafael L.¹

Affiliation:

1. Department of Ophthalmology UT Southwestern Medical Center, Dallas, Texas, United States

2. Department of Ophthalmology UT Southwestern Medical Center, Dallas, Texas, United States 2Department of Ophthalmology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, People's Republic of China

Publisher

Association for Research in Vision and Ophthalmology (ARVO)

Subject

General Medicine

Cited by 30 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Retinal debris triggers cytotoxic damage in cocultivated primary porcine RPE cells;Frontiers in Neuroscience;2024-07-24

2. Complement factor H in molecular regulation of angiogenesis;Medical Review;2024-07-01

3. Modeling complex age-related eye disease;Progress in Retinal and Eye Research;2024-05

4. E3 ubiquitin ligase Herc3 deficiency leads to accumulation of subretinal microglia and retinal neurodegeneration;Scientific Reports;2024-02-06

5. Recent Advances in Our Understanding of Age-Related Macular Degeneration: Mitochondrial Dysfunction, Redox Signaling, and the Complement System;aging and disease;2024