Activation of Cytosolic Calpain, Not Caspase, Is Underlying Mechanism for Hypoxic RGC Damage in Human Retinal Explants-Reference-Cited by-同舟云学术

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Activation of Cytosolic Calpain, Not Caspase, Is Underlying Mechanism for Hypoxic RGC Damage in Human Retinal Explants

Published:2020-11-06 Issue:13 Volume:61 Page:13
ISSN:1552-5783
Container-title:Investigative Opthalmology & Visual Science
language:en
Short-container-title:Invest. Ophthalmol. Vis. Sci.

Author:

Kobayashi-Otsugu Momoko¹²,Orihara Kana¹²,Nakajima Emi¹²,Shearer Thomas R.²,Azuma Mitsuyoshi¹²

Affiliation:

1. Senju Laboratory of Ocular Sciences, Senju Pharmaceutical Corporation Limited, Portland, Oregon, United States

2. Department of Integrative Biomedical & Diagnostic Sciences, Oregon Health & Science University, Portland, Oregon, United States

Publisher

Association for Research in Vision and Ophthalmology (ARVO)

Subject

General Medicine

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. FK962 protects retinal ganglion cell under hypoxia/reoxygenation: possible involvement of glial cell line-derived neurotrophic factor signaling pathway;Experimental Eye Research;2024-09

2. Calpains as mechanistic drivers and therapeutic targets for ocular disease;Trends in Molecular Medicine;2022-08

3. Metabolomic analysis of amino acids and organic acids in aging mouse eyes using gas chromatography–tandem mass spectrometry;Biomedical Chromatography;2022-01-10

4. Inhibition of calpain reduces cell apoptosis by suppressing mitochondrial fission in acute viral myocarditis;Cell Biology and Toxicology;2021-08-08

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