Z<i>nf469</i> Plays a Critical Role in Regulating Synthesis of ECM: A Zebrafish Model of Brittle Cornea Syndrome-Reference-Cited by-同舟云学术

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Znf469 Plays a Critical Role in Regulating Synthesis of ECM: A Zebrafish Model of Brittle Cornea Syndrome

Published:2023-05-31 Issue:5 Volume:64 Page:29
ISSN:1552-5783
Container-title:Investigative Opthalmology & Visual Science
language:en
Short-container-title:Invest. Ophthalmol. Vis. Sci.

Author:

Bao Jing¹²,Yu Xiaoning¹²,Ping Xiyuan¹²,Shentu Xingchao¹²³,Zou Jian¹²⁴⁵

Affiliation:

1. Eye Center, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, Zhejiang, China

2. Zhejiang Provincial Key Lab of Ophthalmology, Hangzhou, Zhejiang, China

3. http://orcid.org/0000-0002-8563-7419

4. The Institute of Translational Medicine, Zhejiang University, Hangzhou, Zhejiang, China

5. https://orcid.org/0000-0002-8884-9978

Publisher

Association for Research in Vision and Ophthalmology (ARVO)

Subject

General Medicine

Reference31 articles.

1. Brittle cornea syndrome: recognition, molecular diagnosis and management;Burkitt Wright;Orphanet J Rare Dis,2013

2. More than meets the eye: expanding and reviewing the clinical and mutational spectrum of brittle cornea syndrome;Dhooge;Human Mutation,2021

3. Evolution of C2H2-zinc finger genes and subfamilies in mammals: species-specific duplication and loss of clusters, genes and effector domains;Tadepally;BMC Evol Biol,2008

4. Common genetic variants near the Brittle Cornea Syndrome locus ZNF469 influence the blinding disease risk factor central corneal thickness;Lu;PLoS Genet,2010

5. New loci associated with central cornea thickness include COL5A1, AKAP13 and AVGR8;Vitart;Hum Mol Genet,2010

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