GSK840 Alleviates Retinal Neuronal Injury by Inhibiting RIPK3/MLKL-Mediated RGC Necroptosis After Ischemia/Reperfusion-Reference-Cited by-同舟云学术

GSK840 Alleviates Retinal Neuronal Injury by Inhibiting RIPK3/MLKL-Mediated RGC Necroptosis After Ischemia/Reperfusion

Published:2023-11-28 Issue:14 Volume:64 Page:42
ISSN:1552-5783
Container-title:Investigative Opthalmology & Visual Science
language:en
Short-container-title:Invest. Ophthalmol. Vis. Sci.

Author:

Feng Yanlin¹,Hu Chenyang¹,Cui Kaixuan¹,Fan Matthew²,Xiang Wu¹,Ye Dan¹,Shi Yuxun¹,Ye Huiwen¹,Bai Xue¹,Wei Yantao¹,Xu Yue¹,Huang Jingjing¹

Affiliation:

1. State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-Sen University, Guangdong Provincial Key Laboratory of Ophthalmology and Visual Science, Guangzhou, China

2. Yale College, Yale University, New Haven, Connecticut, United States

Publisher

Association for Research in Vision and Ophthalmology (ARVO)

Subject

Cardiology and Cardiovascular Medicine

Reference50 articles.

1. Ischemia-reperfusion injury of the retina is linked to necroptosis via the ERK1/2-RIP3 pathway;Gao;Mol Vis,2014

2. Retinal ischemia: mechanisms of damage and potential therapeutic strategies;Osborne;Prog Retin Eye Res,2004

3. Inhibiting multiple forms of cell death optimizes ganglion cells survival after retinal ischemia reperfusion injury;Qin;Cell Death Dis,2022

4. Retinal ganglion cell (RGC) programmed necrosis contributes to ischemia-reperfusion-induced retinal damage;Dvoriantchikova;Exp Eye Res,2014

5. Necroptosis;Linkermann;N Engl J Med,2014

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