Endoplasmic Reticulum Stress Disrupts Mitochondrial Bioenergetics, Dynamics and Causes Corneal Endothelial Cell Apoptosis

Author:

Qureshi Saba1,Lee Stephanie1,Steidl William1,Ritzer Lukas1,Parise Michael2,Chaubal Ananya3,Kumar Varun14

Affiliation:

1. Eye and Vision Research Institute, Department of Ophthalmology, Icahn School of Medicine at Mount Sinai, New York, New York, United States

2. Touro College of Osteopathic Medicine, New York, New York, United States

3. Herricks High School, New Hyde Park, New York, United States

4. Department of Pharmacological Sciences, Icahn School of Medicine at Mount Sinai, New York, New York, United States

Publisher

Association for Research in Vision and Ophthalmology (ARVO)

Subject

Cardiology and Cardiovascular Medicine

Reference75 articles.

1. Proliferative capacity of the corneal endothelium;Joyce;Prog Retin Eye Res,2003

2. Fuchs endothelial corneal dystrophy through the prism of oxidative stress;Jurkunas;Cornea,2018

3. Evidence of oxidative stress in the pathogenesis of Fuchs endothelial corneal dystrophy;Jurkunas;Am J Pathol,2010

4. The role of apoptosis in the pathogenesis of Fuchs endothelial dystrophy of the cornea;Li;Arch Ophthalmol,2001

5. Corneal endothelial cell apoptosis in patients with Fuchs' dystrophy;Borderie;Invest Ophthalmol Vis Sci,2000

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