Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells
Author:
Affiliation:
1. Institute of Molecular Cancer Research, University of Zurich, 8057 Zurich, Switzerland
2. Department of Biochemistry and Molecular Biology, Saint Louis University School of Medicine, St. Louis, MO 63104
Abstract
Publisher
Rockefeller University Press
Subject
Cell Biology
Link
http://rupress.org/jcb/article-pdf/208/5/563/1366785/jcb_201406099.pdf
Reference62 articles.
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2. Nascent chromatin capture proteomics determines chromatin dynamics during DNA replication and identifies unknown fork components;Alabert;Nat. Cell Biol.,2014
3. Replication stress and mechanisms of CNV formation;Arlt;Curr. Opin. Genet. Dev.,2012
4. ATRIP binding to replication protein A-single-stranded DNA promotes ATR-ATRIP localization but is dispensable for Chk1 phosphorylation;Ball;Mol. Biol. Cell.,2005
5. The replication checkpoint protects fork stability by releasing transcribed genes from nuclear pores;Bermejo;Cell.,2011
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