Can ??-Blockers be Used in Diabetic Patients?

Author:

Waal-Manning Hendrika J.

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical)

Cited by 30 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. Effect of Metoprolol and Alprenolol on the Metabolic, Hormonal, and Haemodynamic Response to Insulin-Induced Hypoglycaemia in Hypertensive, Insulin-Dependent Diabetics;Acta Medica Scandinavica;2009-04-24

2. A Cardio-selective Beta Blocker (Metoprolol) in Hypertensive, Insulin-dependent Diabetics;Acta Medica Scandinavica;2009-04-24

3. Modulation of β-adrenergic receptor subtype activities in perioperative medicine: mechanisms and sites of action;British Journal of Anaesthesia;2002-01

4. Beta-blocker therapy for secondary prevention of myocardial infarction in elderly diabetic patients11The analyses upon which this publication is based were performed under Contract No. 500-96-P549, entitled “Utilization and Quality Control Peer Review Organization for the State of Connecticut,” sponsored by the Health Care Financing Administration, Department of Health and Human Services. The content of this publication does not necessarily reflect the views or policies of the Department of Health and Human Services, nor does mention of trade names, commercial products or organizations imply endorsement by the U.S. Government. The authors assume full responsibility for the accuracy and completeness of the ideas presented. This article is a direct result of the Health Care Quality Improvement Program initiated by the Health Care Financing Administration, which has encouraged identification of quality improvement projects derived from analysis of patterns of care, and therefore required no special funding on the part of this Contractor. Ideas and contributions to the author concerning experience in engaging with issues presented are welcomed.;Journal of the American College of Cardiology;1999-11

5. Cardiovascular effects of eating, atenolol and their interaction: β1-adrenergic modulation does not play a predominant role in the genesis of postprandial effects;British Journal of Clinical Pharmacology;1993-11

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