Hypophosphatemia and the Development of Rickets in Osteopetrotic (op/op) Mice
Author:
Publisher
Wiley
Subject
Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism
Reference64 articles.
1. Experimental osteopetrosis;Walker;Clin Orthop,1973
2. The murine mutation osteopetrosis is in the coding region of the macrophage colony stimulating factor gene;Yoshida;Nature,1990
3. Total absence of colony-stimulating factor 1 in the macrophage-deficient osteopetrotic (op/op) mouse;Wiktor-Jedrzejczak;Proc Natl Acad Sci USA,1990
4. Deficiency of osteoclasts in osteopetrotic mice is due to a defect in the local microenvironment provided by osteoblastic cells;Takahashi;Endocrinology,1991
5. Treatment of congenital osteopetrosis in the rabbit with high-dose 1,25-dihydroxyvitamin D;Popoff;J Bone Miner Res,1989
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1. Restriction of Dietary Phosphate Ameliorates Skeletal Abnormalities in a Mouse Model for Craniometaphyseal Dysplasia;Journal of Bone and Mineral Research;2020-07-12
2. Scarcity Despite Wealth: Osteopetrorickets;Journal of Pediatric Endocrinology and Metabolism;2010-01
3. Increased Connective Tissue Extracellular Matrix in the op/op Model of Osteopetrosis;Pathobiology;2009
4. CYP27B1 null mice with LacZreporter gene display no 25-hydroxyvitamin D3-1 -hydroxylase promoter activity in the skin;Proceedings of the National Academy of Sciences;2005-12-21
5. A Characterization of Vitamin D-Independent Intestinal Calcium Absorption in the Osteopetrotic (op/op) Mouse;Archives of Biochemistry and Biophysics;1999-08
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