Mice Lacking Cathepsin K Maintain Bone Remodeling but Develop Bone Fragility Despite High Bone Mass-Reference-Cited by-同舟云学术

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Mice Lacking Cathepsin K Maintain Bone Remodeling but Develop Bone Fragility Despite High Bone Mass

Published:2006-05-08 Issue:6 Volume:21 Page:865-875
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Li Chao Yang,Jepsen Karl J,Majeska Robert J,Zhang Jian,Ni Rujing,Gelb Bruce D,Schaffler Mitchell B

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Reference32 articles.

1. Long-term morbidity and mortality after a clinically diagnosed vertebral fracture in the elderly-a 12- and 22-year follow-up of 257 patients;Hasserius;Calcif Tissue Int,2005

2. Emerging and potential therapies for osteoporosis;Grey;Expert Opin Investig Drugs,2005

3. Interim report and recommendations of the World Health Organization Task-Force for Osteoporosis;Genant;Osteoporos Int,1999

4. Treatment with once-weekly alendronate 70 mg compared with once-weekly risedronate 35 mg in women with postmenopausal osteoporosis: A randomized double-blind study;Rosen;J Bone Miner Res,2005

5. Impaired osteoclastic bone resorption leads to osteopetrosis in cathepsin K-deficient mice;Saftig;Proc Natl Acad Sci USA,1998

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4. Cathepsin K+ Non-Osteoclast Cells in the Skeletal System: Function, Models, Identity, and Therapeutic Implications;Frontiers in Cell and Developmental Biology;2022-07-13

5. A patent review on cathepsin K inhibitors to treat osteoporosis (2011 – 2021);Expert Opinion on Therapeutic Patents;2022-03-10

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