Expression of Measles Virus Nucleocapsid Protein in Osteoclasts Induces Paget's Disease-Like Bone Lesions in Mice-Reference-Cited by-同舟云学术

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Expression of Measles Virus Nucleocapsid Protein in Osteoclasts Induces Paget's Disease-Like Bone Lesions in Mice

Published:2005-11-21 Issue:3 Volume:21 Page:446-455
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Kurihara Noriyoshi,Zhou Hua,Reddy Sakamuri V,Palacios Veronica Garcia,Subler Mark A,Dempster David W,Windle Jolene J,Roodman G David

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Reference41 articles.

1. Genetic linkage of Paget disease of the bone to chromosome 18q;Cody;Am J Hum Genet,1997

2. Paget's disease of bone: Evidence for a susceptibility locus on chromosome 18q and for genetic heterogeneity;Haslam;J Bone Miner Res,1998

3. Genome-wide search in familial Paget disease of bone shows evidence of genetic heterogeneity with candidate loci on chromosomes 2q36, 10p13, and 5q35;Hocking;Am J Hum Genet,2001

4. Paget disease of bone: Mapping of two loci at 5q35-qter and 5q31;Laurin;Am J Hum Genet,2001

5. Linkage of Paget disease of bone to a novel region on human chromosome 18q23;Good;Am J Hum Genet,2002

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1. Osteocytes and Paget’s Disease of Bone;Current Osteoporosis Reports;2024-03-08

2. Roles and mechanisms of optineurin in bone metabolism;Biomedicine & Pharmacotherapy;2024-03

3. Vitamin D and Paget's disease;Feldman and Pike's Vitamin D;2024

4. Osteoclast-derived IGF1 induces RANKL production in osteocytes and contributes to pagetic lesion formation;JCI Insight;2023-07-24

5. A mutation in the ZNF687 gene that is responsible for the severe form of Paget’s disease of bone causes severely altered bone remodeling and promotes hepatocellular carcinoma onset in a knock-in mouse model;Bone Research;2023-03-14

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