Inactivation of Pten in Osteo-Chondroprogenitor Cells Leads to Epiphyseal Growth Plate Abnormalities and Skeletal Overgrowth-Reference-Cited by-同舟云学术

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Inactivation of Pten in Osteo-Chondroprogenitor Cells Leads to Epiphyseal Growth Plate Abnormalities and Skeletal Overgrowth

Published:2007-04-23 Issue:8 Volume:22 Page:1245-1259
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Ford-Hutchinson Alice Fiona,Ali Zenobia,Lines Suzen Elizabeth,Hallgrímsson Benedikt,Boyd Steven Kyle,Jirik Frank Robert

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Reference74 articles.

1. Cellular and molecular interactions regulating skeletogenesis;Colnot;J Cell Biochem,2005

2. Developmental regulation of the growth plate;Kronenberg;Nature,2003

3. Minireview: Transcriptional control of osteoblast differentiation;Karsenty;Endocrinology,2001

4. FGF signaling pathways in endochondral and intramembranous bone development and human genetic disease;Ornitz;Genes Dev,2002

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1. Direct activation of PI3K in osteoblasts and osteocytes strengthens murine bone through sex-specific actions on cortical surfaces;Journal of Bone and Mineral Research;2024-07-03

2. PI3Kα inhibition blocks osteochondroprogenitor specification and the hyper-inflammatory response to prevent heterotopic ossification;2024-07-02

3. Knockdown of Kmt2d leads to growth impairment by activating the Akt/β-catenin signaling pathway;G3: Genes, Genomes, Genetics;2024-01-23

4. Pten knockout in mouse preosteoblasts leads to changes in bone turnover and strength;JBMR Plus;2024-01-04

5. Suppression of Overactive Insulin-Like Growth Factor 1 Attenuates Trauma-Induced Heterotopic Ossification in Mice;The American Journal of Pathology;2023-12

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