SKP2 Contributes to AKT Activation by Ubiquitination Degradation of PHLPP1, Impedes Autophagy, and Facilitates the Survival of Thyroid Carcinoma-Reference-Cited by-同舟云学术

SKP2 Contributes to AKT Activation by Ubiquitination Degradation of PHLPP1, Impedes Autophagy, and Facilitates the Survival of Thyroid Carcinoma

Published:2023-06 Issue:6 Volume:46 Page:360-373
ISSN:1016-8478
Container-title:Molecules and Cells
language:en
Short-container-title:Molecules and Cells

Author:

Shao Yuan^ORCID,Ren Wanli^ORCID,Dai Hao^ORCID,Yang Fangli^ORCID,Li Xiang^ORCID,Zhang Shaoqiang^ORCID,Liu Junsong^ORCID,Yao Xiaobao^ORCID,Zhao Qian^ORCID,Sun Xin^ORCID,Zheng Zhiwei^ORCID,Xu Chongwen^ORCID

Funder

Xi’An Jiaotong University

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,General Medicine

Link

http://pdf.medrang.co.kr/KSMCB/2023/046/KSMCB046-06-06.pdf

Reference55 articles.

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4. The role of Skp2 and its substrate CDKN1B (p27) in colorectal cancer;Bochis;J. Gastrointestin. Liver Dis.,2015

5. SKP2 oncogene is a direct MYC target gene and MYC down-regulates p27KIP1 through SKP2 in human leukemia cells;Bretones;J. Biol. Chem.,2011

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