Apolipoprotein E in Synaptic Plasticity and Alzheimer’s Disease: Potential Cellular and Molecular Mechanisms
Author:
Publisher
Korean Society for Molecular and Cellular Biology
Subject
Cell Biology,Molecular Biology,General Medicine
Link
http://ocean.kisti.re.kr/downfile/crosscheck/ksmcb/JAKO201401458923436.pdf
Reference94 articles.
1. Lack of apolipoprotein E dramatically reduces amyloid β-peptide deposition
2. Differential Effects of ApoE Isoforms on Dendritic Spines In Vivo: Linking an Alzheimer's Disease Risk Factor with Synaptic Alterations
3. Reducing Human Apolipoprotein E Levels Attenuates Age-Dependent A Accumulation in Mutant Human Amyloid Precursor Protein Transgenic Mice
4. Middle-aged human apoE4 targeted-replacement mice show retention deficits on a wide range of spatial memory tasks
5. Balancing Structure and Function at Hippocampal Dendritic Spines
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