Ginsenoside Rg1 promotes β‑amyloid peptide degradation through inhibition of the ERK/PPARγ phosphorylation pathway in an Alzheimer's disease neuronal model-Reference-Cited by-同舟云学术

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Ginsenoside Rg1 promotes β‑amyloid peptide degradation through inhibition of the ERK/PPARγ phosphorylation pathway in an Alzheimer's disease neuronal model

Published:2023-11-23 Issue:1 Volume:27 Page:
ISSN:1792-0981
Container-title:Experimental and Therapeutic Medicine
language:
Short-container-title:Exp Ther Med

Author:

Quan Qiankun¹,Ma Xinxin²,Li Ming¹,Li Xi¹,Yuan Haifeng³

Affiliation:

1. Department of Geriatrics, The Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi 710004, P.R. China

2. Department of Psychology, The Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi 710004, P.R. China

3. Department of Rehabilitation, The Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi 710004, P.R. China

Publisher

Spandidos Publications

Subject

Cancer Research,Immunology and Microbiology (miscellaneous),General Medicine

Reference81 articles.

1. Alzheimer's disease and the amyloid-beta peptide;Murphy;J Alzheimers Dis,2010

2. Amyloid β accelerates phosphorylation of tau and neurofibrillary tangle formation in an amyloid precursor protein and tau double-transgenic mouse model;Seino;J Neurosci Res,2010

3. Synaptic dysfunction in Alzheimer's disease: Mechanisms and therapeutic strategies;Chen;Pharmacol Ther,2019

4. Motor deficits, neuron loss, and reduced anxiety coinciding with axonal degeneration and intraneuronal Aβ aggregation in the 5XFAD mouse model of Alzheimer's disease;Jawhar;Neurobiol Aging,2012

5. Microglia, neuroinflammation, and beta-amyloid protein in Alzheimer's disease;Cai;Int J Neurosci,2014

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